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Photo of Shelby M. Cansler

Shelby M. Cansler

Student Worker

Education/Credentials
  • Bachelor of Arts: Berea College
Research Interests

I study acute and chronic outcomes of traumatic brain injury (TBI) with a particular focus on injury to the optic/visual system. My research focuses on behvaioral and molecular mechanisms in retinal ganglion cells that produce and perpetuate the detrimental pathobiology of TBI.

Contact Information

Peer Reviewed Publications

Hetzer, S.M., Guilhaume-Correa, F., Day, D., Bedolla, A., & Evanson, N.K. 2021. Traumatic optic neuropathy is associated with visual impairment, neurodegeneration, and endoplasmic reticulum stress in adolescent mice. Cells, 10 ,

Cansler, Shelby M; Evanson, Nathan K 2020. Connecting endoplasmic reticulum and oxidative stress to retinal degeneration, TBI, and traumatic optic neuropathy. Journal of neuroscience research, 98 3, 571-574

Guilhaume-Correa, Fernanda; Cansler, Shelby M; Shalosky, Emily M; Goodman, Michael D; Evanson, Nathan K 2020. Greater neurodegeneration and behavioral deficits after single closed head traumatic brain injury in adolescent versus adult male mice. Journal of neuroscience research, 98 3, 557-570

Burke, Emma G; Cansler, Shelby M; Evanson, Nathan K 2018. Indirect traumatic optic neuropathy: modeling optic nerve injury in the context of closed head trauma. Neural regeneration research, 14 4, 593-594

Hetzer, S.M., Shalosky, E. M., Torrens, J., & Evanson, N.K. . Chronic histological outcomes of indirect traumatic optic neuropathy: Persistent degeneration and temporally regulated glial responses. , ,

McGuire, J., Hetzer, S.M, Qu’d, Casagrande, Dobroszi, Burge, Biguma, Bohnert, & Evanson, N.K. . Early measures of TBI severity fail to predict later cognitive impairment in a rat fluid percussion model. , ,