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Photo of Chia-I Ko

Chia-I Ko

Adjunct Assistant Professor

Environmental & Public Health Sciences | College of Medicine

Education/Credentials
  • Doctoral Degree: University of Lorraine, Nancy, France
  • Bachelor's Degree: Taipei Medical University, Taipei city, Taiwan
Contact Information

Peer Reviewed Publications

Ko, Chia-I; Biesiada, Jacek; Zablon, Hesbon A; Zhang, Xiang; Medvedovic, Mario; Puga, Alvaro 2022. The aryl hydrocarbon receptor directs the differentiation of murine progenitor blastomeres. Cell biology and toxicology, ,

Zablon, Hesbon A; Ko, Chia-I; Puga, Alvaro 2021. Converging Roles of the Aryl Hydrocarbon Receptor in Early Embryonic Development, Maintenance of Stemness, and Tissue Repair. Toxicological sciences : an official journal of the Society of Toxicology, 182 1, 1-9

de Gannes, Matthew; Ko, Chia-I; Zhang, Xiang; Biesiada, Jacek; Niu, Liang; Koch, Sheryl E; Medvedovic, Mario; Rubinstein, Jack; Puga, Alvaro 2020. Dioxin Disrupts Dynamic DNA Methylation Patterns in Genes That Govern Cardiomyocyte Maturation. Toxicological sciences : an official journal of the Society of Toxicology, 178 2, 325-337

Ko, Chia-I; Puga, Alvaro 2017. Does the Aryl Hydrocarbon Receptor Regulate Pluripotency? Current opinion in toxicology, 2 , 1-7

Ko, Chia-I; Fan, Yunxia; de Gannes, Matthew; Wang, Qin; Xia, Ying; Puga, Alvaro 2016. Repression of the Aryl Hydrocarbon Receptor Is Required to Maintain Mitotic Progression and Prevent Loss of Pluripotency of Embryonic Stem Cells. Stem cells (Dayton, Ohio), 34 12, 2825-2839

Wang, Qin; Kurita, Hisaka; Carreira, Vinicius; Ko, Chia-I; Fan, Yunxia; Zhang, Xiang; Biesiada, Jacek; Medvedovic, Mario; Puga, Alvaro 2016. Ah Receptor Activation by Dioxin Disrupts Activin, BMP, and WNT Signals During the Early Differentiation of Mouse Embryonic Stem Cells and Inhibits Cardiomyocyte Functions. Toxicological sciences : an official journal of the Society of Toxicology, 149 2, 346-57

Carreira, Vinicius S; Fan, Yunxia; Kurita, Hisaka; Wang, Qin; Ko, Chia-I; Naticchioni, Mindi; Jiang, Min; Koch, Sheryl; Zhang, Xiang; Biesiada, Jacek; Medvedovic, Mario; Xia, Ying; Rubinstein, Jack; Puga, Alvaro 2015. Disruption of Ah Receptor Signaling during Mouse Development Leads to Abnormal Cardiac Structure and Function in the Adult. PloS one, 10 11, e0142440

Carreira, Vinicius S; Fan, Yunxia; Wang, Qing; Zhang, Xiang; Kurita, Hisaka; Ko, Chia-I; Naticchioni, Mindi; Jiang, Min; Koch, Sheryl; Medvedovic, Mario; Xia, Ying; Rubinstein, Jack; Puga, Alvaro 2015. Ah Receptor Signaling Controls the Expression of Cardiac Development and Homeostasis Genes. Toxicological sciences : an official journal of the Society of Toxicology, 147 2, 425-35

Winans, Bethany; Nagari, Anusha; Chae, Minho; Post, Christina M; Ko, Chia-I; Puga, Alvaro; Kraus, W Lee; Lawrence, B Paige 2015. Linking the aryl hydrocarbon receptor with altered DNA methylation patterns and developmentally induced aberrant antiviral CD8+ T cell responses. Journal of immunology (Baltimore, Md. : 1950), 194 9, 4446-57

Ko, Chia-I; Wang, Qin; Fan, Yunxia; Xia, Ying; Puga, Alvaro 2014. Pluripotency factors and Polycomb Group proteins repress aryl hydrocarbon receptor expression in murine embryonic stem cells. Stem cell research, 12 1, 296-308

Wang, Qin; Chen, Jing; Ko, Chia-I; Fan, Yunxia; Carreira, Vinicius; Chen, Yinglei; Xia, Ying; Medvedovic, Mario; Puga, Alvaro 2013. Disruption of aryl hydrocarbon receptor homeostatic levels during embryonic stem cell differentiation alters expression of homeobox transcription factors that control cardiomyogenesis. Environmental health perspectives, 121 11-12, 1334-43

Wang, Jingcai; Chen, Liang; Ko, Chia-I; Zhang, Lin; Puga, Alvaro; Xia, Ying 2012. Distinct signaling properties of mitogen-activated protein kinase kinases 4 (MKK4) and 7 (MKK7) in embryonic stem cell (ESC) differentiation. The Journal of biological chemistry, 287 4, 2787-97

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Department of Environmental & Public Health Sciences
Division of Environmental Genetics and Molecular Toxicology
Kettering Lab Building
Room 133
160 Panzeca Way
Cincinnati, OH 45267-0056
Mail Location: 0056

Phone: 513-558-5704